Original articleA molecular and serologic evaluation of enteroviral involvement in human myocarditis
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2016, Cardiovascular Pathology: Fourth EditionMiR-342-5p suppresses coxsackievirus B3 biosynthesis by targeting the 2C-coding region
2012, Antiviral ResearchCitation Excerpt :Severe health consequences have been associated with outbreaks of coxsackievirus infection (Cui et al., 2010a; Verma et al., 2009; Wong et al., 2011). Moreover, group B coxsackievirus (CVB) infection has been implicated in the development of myocarditis and dilated cardiomyopathy (Bowles et al., 1986; Jin et al., 1990; Knowlton, 2008; Martin et al., 1994; Tracy et al., 1990). In spite of this, there is presently no specific and effective therapy available for CVB infection.
Patchy myocardial pattern of virus sequence persistence in heart transplant recipients - Possible role of sampling error in the etiology
2011, Transplantation ProceedingsCitation Excerpt :Kühl et al11 showed that adenovirus persistence worsened the prognosis of idiopathic left ventricle failure. Recent results have shown that virus eradication with interferon may attenuate the clinical consequences of DCM due to an entero- or adenovirus etiology.7–9 The effects of interferon therapy in acute myocarditis-related end stage heart failure, however remain controversial.19
Coxsackievirus B<inf>3</inf> affects endothelial tight junctions: Possible relationship to ZO-1 and F-actin, as well as p38 MAPK activity
2007, Cell Biology InternationalCitation Excerpt :Group B coxsackievirus (CVB) consists of six serotypes, of which CVB3 in particular is considered to be the most frequent cause of human viral heart disease as well as other related disorders (Tracy et al., 1990; Baboonian et al., 1997; Pallansch, 1997).
PKR's protective role in viral myocarditis
2003, Virology