Short communicationCell killing by ultraviolet-inactivated human immunodeficiency virus
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Inflammasomes in T cells
2022, Journal of Molecular BiologyCitation Excerpt :The other was a lytic form of cell death that was preceeded by cell swelling. The identification of the latter as a form of programmed cell death, dates back to a time when any programmed cell death was equated with apoptosis.83 The analysis of ex vivo specimen of lymph nodes from infected individuals suggested that the cell death triggered by HIV occurs primarily in non-productively infected bystander cells (not staining positive for viral RNA) rather than in productively infected cells.84
Possible Biomarkers for the Early Detection of HIV-associated Heart Diseases: A Proteomics and Bioinformatics Prediction
2015, Computational and Structural Biotechnology JournalCitation Excerpt :We also could not conduct proteomics studies directly in HIV-infected individuals without first identifying protein profiles of cells infected by HIV alone, because most HIV-patients are co-infected with other pathogenic viruses and microorganisms that may produce factors similar to those induced by HIV. We therefore chose a genetically stable, single-cell-clone of a human T-cell line (RH9) [55] and infected it with a biologically cloned North American HIV-Clade B (HBX) in vitro. The differentially-regulated proteins expressed in HIV-infected and counterpart uninfected cells were evaluated at numerous time points from 1.5-, 3,6-, 12- and 24-h, followed by weekly, then 4–6 weeks samplings for 6–7 months post-infection.
Ultraviolet irradiation and cytokines as regulators of HIV latency and expression
1994, Chemico-Biological Interactions