Trends in Genetics
Volume 5, 1989, Pages 326-331
Human telomeres: fusion and interstitial sites
References (40)
Cell
(1987)- et al.
Cell
(1985) - et al.
Cell
(1988) Cancer Genet. Cytogenet.
(1988)- et al.
Cell
(1986) - et al.
Cell
(1984) Yeast
(1987)- et al.
Nucleic Acids Res.
(1987) - et al.
Mol. Cell. Biol.
(1989) - et al.
Cell
(1989)
Nature
(1989)
Nature
(1984)
Nature
(1989)
Mol. Cell. Biol.
(1988)
Nature
(1988)
Nucleic Acids Res.
(1989)
Nature
(1989)
Nature
(1989)
Nature
(1988)
Cited by (248)
Expansion of Interstitial Telomeric Sequences in Yeast
2015, Cell ReportsCitation Excerpt :They are believed to result from the insertions of telomeric repeats during the repair of double-stranded DNA breaks via non-homologous end-joining (NHEJ) (Azzalin et al., 2001; Nergadze et al., 2004), possibly involving telomerase (Nergadze et al., 2007). Like many other microsatellites, s-ITSs are polymorphic in length (Hastie and Allshire, 1989); for instance, their significant length polymorphism has been observed in gastric tumors (Mondello et al., 2000). Cytogenetic analysis has co-localized ITSs with spontaneous and induced chromosome breakage sites in primates (Ruiz-Herrera et al., 2005) and rodents (Musio et al., 1996).
Effect of bleomycin on interstitial telomeric sequences of immortalized Chinese hamster ovary cells
2009, Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis
- ∗
R.C. Allshire is now at Cold Spring Harbor Laboratory, PO Box 100, Cold Spring Harbor, NY 11724, USA.
Copyright © 1989 Published by Elsevier Ltd.