Serum levels of β-carotene and other carotenoids in Parkinson's disease
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Cited by (33)
Targeting urate to reduce oxidative stress in Parkinson disease
2017, Experimental NeurologyCitation Excerpt :On the contrary, another study of 11 patients found a small reduction (19%) in the activity of GPx in the SN in patients with PD (Kish et al., 1985). Although reduced levels of antioxidants vitamin A, C and E and reduced activities of SOD and glutathione reductase have been reported in AD (Rinaldi et al., 2003; Lovell et al., 1995), most early studies of vitamin A, C and E and beta-carotene found no association with PD (King et al., 1992; Fernandez-Calle et al., 1993; Jimenez-Jimenez et al., 1993; Fernandez-Calle et al., 1992; Jimenez-Jimenez et al., 1992). Abbott et al. found lower plasma levels of vitamins E and A in PD cases than in controls (Abbott et al., 1992).
Nutrigenetic Effect on Intestinal Absorption of Fat-Soluble Microconstituents (Vitamins A, E, D and K, Carotenoids and Phytosterols)
2009, Global Issues in Food Science and TechnologyGenes and intestinal absorption of lipidic microconstituents (fat-soluble vitamins, carotenoids and phytosterols)
2009, Cahiers de Nutrition et de DietetiqueEvidence of increased reactive species formation by retinol, but not retinoic acid, in PC12 cells
2008, Toxicology in VitroCitation Excerpt :The ability of retinol and related molecules to act as antioxidant or protective agents in biological systems has been strongly supported in recent years. Several studies have reported a correlation between low serum levels of retinoids, or carotenoids, and the incidence of pathologies that are related in part to increased ROS production, such as neurodegenerative diseases and neoplasias (Jimenez-Jimenez et al., 1993; Greenwald et al., 2006). The mechanisms underlying these apparent protective properties are still not well understood.
Redox status of plasma coenzyme Q10 indicates elevated systemic oxidative stress in Parkinson's disease
2004, Journal of the Neurological Sciences