Effects of noise on cochlear potentials and endolymph potassium concentration recorded with potassium-selective electrodes
References (41)
- et al.
Effects of exposure to noise on ion transport in guinea pig cochlea
Hearing Res.
(1979) Acoustic trauma in the guinea pig I. Electrophysiology and histology
Acta Oto-Laryngol.
(1965)- et al.
Temporary threshold shifts in chinchilla: Electrophysiological correlates
J. Acoust. Soc. Am.
(1972) Ethacrynic acid ototoxicity as a general model in cochlear pathology
Adv. Oto-Rhino-Laryngol.
(1977)- et al.
Temperature coefficients of cochlear potentials
Am. J. Physiol.
(1960) - et al.
Modification of cochlear potentials by streptomycin poisoning and by extensive venous obstruction
Laryngoscope
(1958) Effect of temperature on cochlear responses during and after exposure to noise
J. Acoust. Soc. Am.
(1976)- et al.
Acoustic trauma following intermittent exposure to tones
Ann. Otol. Rhinol. Laryngol.
(1959)
Factor analysis of cochlear injuries and changes in electrophysiological potentials following acoustic trauma in the guinea pig
J. Acoust. Soc. Am.
Physicochemical properties of a liquid ion exchanger microelectrode and its application to biological fluids
Jap. J. Physiol.
The role of vasoconstriction in noise induced hearing loss
Ann. Otol. Rhinol. Laryngol.
Dependence of the cochlear microphonics and the summating potential on the endocochlear potential
J. Acoust. Soc. Am.
Membrane resistance in endolymphatic walls of the first turn of the guinea pig cochlea
J. Acoust. Soc. Am.
Dynamic changes in cochlear potentials and endolymph concentrations
J. Otolaryngol. Soc. Aust.
Effect of chemical alteration in the endolymph on the cochlear potentials
Acta. Oto-Laryngol.
Negative potential in scala media during early stage of anoxia
Acta Oto-Laryngol.
Ion transport in the guinea pig cochlea I. Sodium and potassium transport
Acta Oto-Laryngol.
Some observations of negative endocochlear potential during anoxia
Acta Oto-Laryngol.
Cited by (44)
The aging cochlea: Towards unraveling the functional contributions of strial dysfunction and synaptopathy
2019, Hearing ResearchCitation Excerpt :On the other hand, the intrastrial space already maintains a high positive potential, suggesting that the EP is generated by intermediate and basal cells (Nin et al., 2008; Salt et al., 1987) (Fig. 1B). Furthermore, following pharmacological blocking of the NKCC1 cotransporter or anoxia, the EP dropped much more rapidly and recovered faster than the [K+e] (Rybak and Morizono, 1982; Salt and Konishi, 1979). Below, we first briefly review the literature describing age-related histopathologies in the different cell types of the stria vascularis (Table 1).
A behavioral measure of the cochlear changes underlying temporary threshold shifts
2011, Hearing ResearchCitation Excerpt :The OHC electromotile mechanism is physiologically vulnerable (Johnstone et al., 1986; Ruggero and Rich, 1991; Sellick et al., 1982) and its dysfunction can account for the raised thresholds, recruitment, and loss of frequency selectivity observed in permanent sensorineural hearing loss (Moore, 2007). OHC dysfunction has also been suggested as a cause of moderate fatigue following noise exposure on the basis of physiological studies in non-human animals (Cody and Russell, 1988; Johnstone et al., 1989; Salt and Konishi, 1979), and psychoacoustic (Norton and Mott, 1987; Patuzzi, 1998a) and objective (Kim et al., 2005; Vinck et al., 1999) measures in humans. However, previous studies have not determined if OHC dysfunction is the exclusive cause of fatigue or if dysfunction of IHCs also contributes.
Static material properties of the tectorial membrane: A summary
2003, Hearing Research