Brief communicationOxidative damage caused by free radicals produced during catecholamine autoxidation: Protective effects of O-methylation and melatonin
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2023, Neurobiology of DiseaseCitation Excerpt :Beyond serving as a PD marker, in general, DAT and vesicular monoamine transporter 2 (VMAT2) in the CNS collectively regulate dopamine homeostasis. Free extracellular dopamine quickly oxidizes and forms dopamine radicals such as quinolones, damaging dopamine neurons (Anderson et al., 2011; Miller et al., 1996; Terland et al., 1997). DAT rapidly clears extracellular dopamine and transports it into the intracellular space for repackaging into synaptic vesicles via VMAT2 (Cheng et al., 2015; Pramod et al., 2013; Vaughan and Foster, 2013).
Carving the senescent phenotype by the chemical reactivity of catecholamines: An integrative review
2022, Ageing Research ReviewsCitation Excerpt :The propensity for autoxidation, which is the common chemical feature of any catechols (Fig. 4), may be neutralized metabolically by their enzymatic methylation (Meiser et al., 2013). CA autoxidation has been thoroughly studied because of its importance for heart and brain pathology (Bindoli et al., 1992; Bisaglia et al., 2014; Cavalieri et al., 2002; Delcambre et al., 2016; El-Ayaan et al., 1997; Miller et al., 1996; Monzani et al., 2019; Singal et al., 1983; Sulzer and Zecca, 1999; Umek et al., 2018). In neurons, CA autoxidation is initiated by superoxide or ferric iron.
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2021, Neurochemistry InternationalDoes membrane curvature elastic energy play a role in mediating oxidative stress in lipid membranes?
2021, Free Radical Biology and MedicineCitation Excerpt :LOXes have been implicated in a large range of age-related diseases [64,65], since overexpression or upregulation of LOXes can lead to high levels of oxidative stress in cells [66–68]. ROS are produced in vivo via enzymatic oxidation reactions by enzymes like mono amine oxidase (MAO) [69] in the mitochondria, peroxisomes and endoplasmic reticula [5,70], or through autooxidation of compounds like catecholamines, hydroquinone and sterols [56,71]. Exogenous stimuli like ionising radiation [72], UV light, alcohol and tobacco smoke are also sources of in vivo ROS production [1,73–75].