The Journal of Steroid Biochemistry and Molecular Biology
Steroids and opioid receptors
References (63)
- et al.
Modulation of the binding characteristics of hypothalamic mu opioid receptors in rats by gonadal steroids
J. Steroid Biochem. Molec. Biol.
(1991) - et al.
Effects of in vivo estradiol and progesterone on tritiated flunitrazepam binding in rat spinal cord
Neuroscience
(1988) Rapid membrane effects of steroids hormones: an emerging concept in neuroendocrinology
Trends Neurosci.
(1990)Non-genomic and genomic effects of steroids on neuronal activity
Trends Pharmac. Sci.
(1991)- et al.
Anatomy of CNS opioid receptors
Trends Neurosci.
(1988) A simple computer program for Scatchard plot analysis of steroid receptors including non-specific binding correction on a low cost desk top calculator
J. Steroid Biochem.
(1979)- et al.
Interference of danazol with the radioimmunoassay of steroid hormones
J. Steroid Biochem.
(1982) - et al.
Steroid binding to synaptic plasma membrane: Differential binding of glucocorticoids and gonadal steroids
J. Steroid Biochem.
(1983) - et al.
Rapid conversion of high into low striatal D2-dopamine receptor agonist binding state after an acute physiological dose of 17beta-estradiol
Neurosci. Lett.
(1988) - et al.
Progesterone derivatives that bind to the digitalis receptor
Trends Pharmac. Sci.
(1989)
Neurosteroids: a new function in the brain
Biol. Cell. (Paris)
Specific and non-specific physicochemical interactions of glucocorticoids and related steroids with rat thymus cells in vitro
J. Biol. Chem.
Transcriptional control by nuclear receptors
FASEB J.
Steroid hormones: Humoral signals which alter brain cell properties and functions
Rec. Prog. Horm. Res.
Action of estrogens and progestins on nerve cells
Science
Steroid hormone metabolites are barbiturate-like modulators of the GABA receptor
Science
Pharmacology of opioids
Pharmac. Rev.
Classification of opioid receptors
Br. Med. Bull.
Simultaneous analysis of families of sigmoidal curves: application to bioassay, radioligand assay, and physiological dose-response curves
Am. J. Physiol.
The discrimination between different blood-CSF barrier dysfunctions and inflammatory reactions of the CNS by a recent evaluation graph for the protein profile of cerebrospinal fluid
J. Neurol.
Hormone binding globulin levels in patients with hereditary angiooedema during treatment with Danazol
Clin. Endocr. (Oxf.)
Demonstration of sex hormone binding globulin in human cerebrospinal fluid
Clin. Endocr. (Oxf.)
Protein binding of cortisol in human cerebrospinal fluid
J. Clin. Endocr. Metab.
Strategy and organization of intralaboratory radioimmunoassay quality control. Invited review paper
The blood-brain barrier, a dynamic regulatory interface
Molec. Physiol.
Protein size and cerebrospinal fluid composition
Klin. Wschr.
Neuroendocrinology of cerebrospinal fluid: Peptides, steroids, and other hormones
Neurosurgery
Passage of proteins from blood to cerebrospinal fluid. Model for transfer by pores and vesicles
Receptor-mediated peptide transport through the blood-brain barrier
Endocrine Rev.
Transcytotic pathway for blood-borne proteins through the blood-brain barrier
Cited by (44)
Amphetamine withdrawal differentially affects hippocampal and peripheral corticosterone levels in response to stress
2016, Brain ResearchCitation Excerpt :In support of this hypothesis, methamphetamine exposure and withdrawal has been reported to increase the permeability of the blood brain barrier to otherwise impermeable binding proteins (Kiyatkin and Sharma, 2012; Sharma and Ali, 2006). Furthermore, failure to show differences in bound steroid in the plasma, as in the current study, does not preclude increased binding protein in the cerebrospinal fluid (Schwarz and Pohl, 1994). It is therefore conceivable that chronic amphetamine exposure and withdrawal increases the permeability of the blood brain barrier to binding proteins such as CBG, which is known to play a critical role in maintaining a corticosterone pool accessible to central tissues following stress (Mattos et al., 2013).
Testosterone in the brain: Neuroimaging findings and the potential role for neuropsychopharmacology
2013, European NeuropsychopharmacologyCitation Excerpt :Additionally it has to be considered that the blood brain barrier is easily crossable for steroids due to their high lipid solubility. Correlations between free serum levels of DHEAS, androstendione and testosterone and levels measured within the cerebrospinal fluid (CSF) have been found (Schwarz and Pohl, 1992, 1994). Thus, testosterone has shown to act via nongenomic mechanisms in the brain (e.g., modulation of opioid receptors) (Schwarz et al., 1989).
Endocrine disruption via estrogen receptors that participate in nongenomic signaling pathways
2011, Journal of Steroid Biochemistry and Molecular BiologyCitation Excerpt :These other physiologic estrogens have long been labeled weak estrogens because they were tested exclusively via the genomic signaling pathway. Now we find that some of them (that have so far been tested) are actually quite potent via the nongenomic signaling pathway [29–32]. Their ability to act potently may relate to actions at particular life stages of women in which these hormones are quite prominent.
Sex differences in pain and analgesia: The role of gonadal hormones
2004, European Journal of PainEffects of perinatal buprenorphine and methadone exposures on striatal cholinergic ontogeny
2002, Neurotoxicology and Teratology