Elsevier

The Lancet

Volume 273, Issue 7084, 6 June 1959, Pages 1180-1182
The Lancet

ORIGINAL ARTICLES
THE PSYCHIATRIC COMPLICATIONS OF TOXOPLASMOSIS

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Cited by (22)

  • Childhood infection and subsequent risk of psychotic disorders in adults: A systematic review and meta-analysis

    2020, Asian Journal of Psychiatry
    Citation Excerpt :

    However, few studies have assessed the association between infection in early life and later psychosis. Several reports of psychosis following exposure to infections such as toxoplasmosis (Minto and Roberts, 1959), influenza (Chang et al., 2015), and herpes virus (Schlitt et al., 1985) have been published, implicating microbial pathogens in this disorder. Epidemiological studies (Benros et al., 2016; Blomström et al., 2014; Dalman et al., 2008; Debost et al., 2019; Khandaker et al., 2018, 2015; Köhler-Forsberg et al., 2019; Koponen et al., 2004; Leask et al., 2002; Liang and Chikritzhs, 2012; Nielsen et al., 2014; Rantakallio et al., 1997; Weiser et al., 2010) of the association between childhood infection and the risk of subsequent psychosis have reported inconsistent findings.

  • Decreased level of psychobiological factor novelty seeking and lower intelligence in men latently infected with the protozoan parasite Toxoplasma gondii Dopamine, a missing link between schizophrenia and toxoplasmosis?

    2003, Biological Psychology
    Citation Excerpt :

    It has recently been shown that contact with cats is also an important risk factor in schizophrenia (Torrey and Yolken 1995; Torrey et al., 2000; Torrey and Yolken, 2002). Many studies have also directly demonstrated frequent association between acute toxoplasmosis and schizophrenia or higher prevalence of latent toxoplasmosis in patients with schizophrenia than in the normal population (Minto and Roberts, 1959; Robertson, 1965; Kramer, 1966; Ladee et al., 1966; Yolken et al., 2001). These results, as well as our indirect indices for increased level of dopamine in the brain of infected subjects, suggest that T. gondii can be an etiological agent in at least some forms of schizophrenia.

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