Journal of Biological Chemistry
Volume 288, Issue 2, 11 January 2013, Pages 1295-1306
Molecular Bases of DiseaseInhibition of Glycogen Synthase Kinase-3 Ameliorates β-Amyloid Pathology and Restores Lysosomal Acidification and Mammalian Target of Rapamycin Activity in the Alzheimer Disease Mouse Model: IN VIVO AND IN VITRO STUDIES*
Under a Creative Commons license
open access
Alzheimer Disease
Amyloid
Cell Signaling
Drug Action
Glycogen Synthase Kinase 3
Lysosomal Acidification
Neurodegenerative Diseases
Presenilin
S6 Kinase
mTOR
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This work was supported by Israel Science Foundation Grant 341/10 and Seventh Framework Program European Union Grant 223276 “NeuroGSK3” (to H. E.-F.) and by Alzheimer's Association Grant NIRG-11-205535 (to D. F.).
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These authors contributed equally to this work. Submitted in partial fulfillment of the requirements for a Ph.D. degree at Tel Aviv University.
© 2013 ASBMB. Currently published by Elsevier Inc; originally published by American Society for Biochemistry and Molecular Biology.